Page24

Update item information
Title Exploring human heredity
Subject Heredity, Human; Medical genetics; Adaptation (Biology)
Description Twentieth Annual Frederick William Reynolds Lecture.
Creator Stephens, Fayette Ellsworth, 1890-
Publisher Extension Division, University of Utah
Date 1956-01-16
Date Digital 2008-05-29
Type Text
Format image/jpeg
Digitization Specifications Original scanned on Epson Expression 10000XL flatbed scanner and saved as 400 ppi uncompressed tiff. Display images generated in PhotoshopCS and uploaded into CONTENTdm Aquisition Station.
Resource Identifier http://content.lib.utah.edu/u?/reynolds,362
Source LD5526 .U8 n.s. v.47 no.11
Language eng
Relation Digital reproduction of "Exploring human heredity" J. Willard Marriott Library Special Collections
Rights Digital Image Copyright University of Utah
Metadata Cataloger Seungkeol Choe; Ken Rockwell
ARK ark:/87278/s64q7rxv
Setname uu_fwrl
Date Created 2008-07-29
Date Modified 2008-07-31
ID 319670
Reference URL https://collections.lib.utah.edu/ark:/87278/s64q7rxv

Page Metadata

Title Page24
Description 24 TWENTIETH ANNUAL REYNOLDS LECTURE Figure 10 Sometimes apparently identical or similar traits are caused by different genes acting in quite different ways. Hemophilia and telangiectasia are hereditary traits characterized by excessive bleeding. Hemophilia is caused by a recessive sex-linked gene which results in a defect of the blood that prolongs the clotting time. On the other hand, telangiectasia is due to an autosomal dominant gene which produces a defect in the walls of the blood vessels, also resulting in bleeding. Figure 10 shows a man with telangiectasia. The small red spots are characteristic of this disease. The genetic picture of these two traits is quite different. Hemophilia, with a very few exceptions, occurs only in males and is transmitted by females who themselves are not bleeders. Telangiectasia, on the other hand, occurs as often in women as in men. It never occurs in a child unless it also occurs in one of the parents. Approximately one-half of the children of affected parents are bleeders. The normal children of an affected parent have all normal offspring. The effects of genes become manifest at different times in the lives of individuals. Some hereditary traits are congenital, or present i
Format image/jpeg
Identifier 024-RNLT-StephensF_Page24.jpg
Source Original Manuscript:Exploring human heredity by Fayette E. Stephens.
Setname uu_fwrl
Date Created 2008-07-29
Date Modified 2008-07-29
ID 319658
Reference URL https://collections.lib.utah.edu/ark:/87278/s64q7rxv/319658