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Show 310 / Bibliography upon the EEG of acute injury to the brain stem activating system. Electroencephclin.Neurophysiol. 1:475-486. [Decerebrate cats: lesions affecting the ascending reticular system; see Chapter 9.) * * Lindsley,DB & Sassman,WH (1938) Autonomic activity and brain potentials associated with "voluntary" control of the pilomotors (mm. arrectores pilorum). l.Neurophysiol. 1:342-349. (Motion pictures of piloerection and pupillodilation; see Chapter 13.] Lindsley,DB; Schreiner,LH; Knowles,WB & Magoun,HW (1950) Behavioral and EEG changes following chronic brain stem lesions in the cat. Electroencephclin.Neurophysiol 2:483-(?). [Lesions to reticular brain stem core caused permanent somnolence; see Chapter 9.] * Linksz,A (1931) Der Einfluss der Sympathicusausschaltung auf die Blut-Kammerwasserschranke. Klin. Wschr. 10:839-840. (Sympathectomy caused slight increase, paracentesis or mustard oil great increase of protein leakage into the aqueous; see Chapters 6 and 11.] * Linksz,A (1931) Eine neue Methode zur exakten quantitativen Bestimmung des Fluoreszeins in iiusserst schwachen Konzentrationen. Arch. Augenheilk 104:470489. • Linksz,A (1931) Untersuchungen iiber den Einfluss des Sympathikus auf die Permeabilitiit der Augengefiisse. (Zugleich ein Beitrag zur Lehre vom vegetativen Nervensystem.) ArchAugenheilk 104:264-323. (After sympathectomy in rabbits, increased protein entered the aqueous; after postganglionic sympathetic lesions, the effect gradually reversed. Also saw miosis upon pucturing the anterior chamber; see Chapters 6 and 11.] * * Linksz,A (1932) Ueber Glaukomtherapie und ihre Beziehungen zum vegetativen Nervensystem. Ber.49. Zus.dtschophthaLGes.,Leipzig, 1932. A Wagemann, Ed.;·Miinchen: J.F. Bergmann. [See Chapters 11 and 40.] Linksz,A (1938) Zur Frage des sog. trophischen Einflusses des Sympathikus. Z.Biol. 99:182-185. (Acute sympathectomy increased protein-passage into the anterior chamber; chronic sympathectomy reversed this, while pupillary effect of adrenaline also reversed.) * van Lint,-.: see Van Lint. Ma/ Lippman,JI (1982) Pupillary abnormalities associated with posterior chamber Jens implantation. Ophthalmic Surg. 13:197-200. • Lippmann,O (1944) Paralysis of divergence due to cerebellar tumor. Arch Ophthal, Chicago 31:299-301. (Pupils remained normal.] McV Lippman,W (1971) Potentiation of ocular response to adrenaline by 3,3-dimethyl-1-(3-methylaminopropyl)-1phenylphthalan (Lu 3-010) and a thiophthalan analogue (Lu 5-003): blockers of catecholamine uptake. Experientia 27:927-928. [According to MEDLAR, pupillary effects (cocaine-like) included; see Chapter 14.] MaV Lippman,W & Pugsley,TA (1976) Effects of viloxazine, an antidepressant agent, on biogenic amine uptake mechanisms and related activities. [According to MEDLAR, potentiation of ocular effects of /-adrenaline in the rabbit; see Chapter 14.] +/ Lips,U; Conrad,!; Zevouno,F & Schappler-Scheele,B (1982) Bericht iiber 2 Fiille eines irreversiblen HornerSyndroms nach Punktion der Vena jugularis interna. Anlisth.lntensivther.Notfallmed.,Stuttgart 17:301-(?). [Cited by H.S. Thompson in 1984 literature review. *p Lipschitz,R (1906) Beitriige zur Lehre von der Facialisliihmung nebst Bemerkungen zur Frage der Nervenregeneration. Mschr.Psychiat.Neurol. 20:84-167. (Misdirection dyskinesia; see Chapter 11.] • Lipschiitz,H & Schilf,E (1931) Ueber die Wirkung von Acetylcholin auf die Iris des Warmbliiters. Archexper. PathPharmacoL 162:617-622. • Lipton,S (1968) Percutaneous electrical cordotomy in relief of intractable pain. Brit.med.I. 2:210-212. (52 patients; Homer's syndrome mentioned but did not see it himself.] * Lissak,K (1939) Liberation of acetylcholine and adrenaline by stimulating isolated nerves. Amer.I. PhysioL 127:263-271. [No pupil; obtained adrenalinelike substance from in vitro adrenergic nerves; see Chapters 6, 11, and 14.] • List,CF & Peet,MM (1938) Sweat secretion in man. IV . Sweat secretion of the face and its disturbances. Arch NeuroLPsychiat., Chicago 40:443-4 70. [No pupil; see Chapters 6 and 25.] * Lister,A (1934) Tonic pupils. Proc.roy.Soc.Med. 27: 1057-. Also in Amer.I. Ophthal. 17:857. • Lister,AEJ (1931) Discussion of papers by R.F. Moore and G. Holmes. Trans.ophthal.Soc.U.K 51:226-. (Tonic pupil; own patient was in "apparently perfect health.") * Lister,] (1853) Observations on the contractile tissue of Linn,ML & Jones,LT (1968) Rate of lacrimal excretion of ophthalmic vehicles. Amer.l.Ophthal. 65:76-78. (ver. IM, 1968, 9:7856.) (Cited by Hardberger et al., 1980, and by Mishima, 1981; see Chapter 14.] V Linner,E & Prijot, E (1955) Cervical sympathetic ganglionectomy and aqueous flow. Arch.Ophthal.,Chicago 54:831-833. [Cited by Casey, 1966; see Chapter 6.] * Linquette,H; Fossati,P; Luez,G; Decoul,M & Woillez,M (1964) Signe d'Argyll-Robertson d'etiologie non-specifique au cours d'une neuropathie dibetique. (Ass.Diabet.Lang.fr., 24 Oct.) Presse med. 72:53,3225. V Linser,P (1903) Ueber juvenile Tabes und ihre Beziehungen zur hereditiiren Syphilis. Munchmed. Wschr. 50:637-638. rPupils unequal, fixed, or Argyll Robertson.] v. Linstow,O (1867) Bemerkungen iiber die wiihrend der Jahre 1845 bis 1865 in der lrrenanstalt bei Schleswig beobachteten Fiille von Paresis universalis progressiva. Allg.Z.Psychiat. 24:436-463. (Pupils p. 440: miosis, mydriasis, sluggish or fixed; one-third of patients had defective pupils.] |
