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Show 140 LITERATURE ABSTRACTS ment ischemia was evaluated with clinical examination and fluorescein angiogram of the iris. Exhaustive (and exhausting) tables and descriptions of results are presented, but the last several pages of "comments" nicely summarize the not-surprising results and their relationship to presumed routes of blood supply to the anterior segment in humans. Lyn A. Sedwick, M.D. Anterior Ischemic Optic Neuropathy: VIII. Clinical Features and Pathogenesis of Post-hemorrhagic Amaurosis. Hayreh SS. Ophthalmology 1987;94:1488-502 (Nov). [Reprint requests to Dr. S. S. Hayreh, Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City, lA 52242.] Dr. Hayreh proposes "(1) to give a comprehensive clinical picture of this disease, based on an exhaustive review of the literature and [his] own experience; and (2) to discuss its pathogenesis in the light of current knowledge." He admirably succeeds. Four patients are presented in detail, with optic nerve photographs and fluorescein angiography, who were seen when in the acute phase of visual loss following significant systemic hemorrhage. The literature is extensively reviewed back to the case documented by Hippocrates, and conflicting descriptions and reports of this disorder are renconciled logically. Perhaps the best section of the paper is that regarding pathogenesis. This is an excellent and thoughtful paper. LYIl A. Sedwick, M.D. Chiasmal Neuropathy Secondary to Rheumatoid Pachymeningitis. Weinstein GW, Powell SR, Thrush WP. Am J Ophthalmol 1987;104:439-40 (Oct) [Inquiries to Dr. G. W. Weinstein, West Virginia University Medical Center, Morgantown, WV 26506.] A 42-year-old man with rheumatoid arthritis reported gradual visual loss in the right eye. He had "hand motions" right eye and a superotemporal visual field cut in his left eye. Computerized tomographic scanning of the sella demonstrated a mass that was felt by biopsy and cerebrospinal fluid an"I,·,;" tr> f"!'rpsen t rheumatoid pachymenin- J elm NCllru-vphlhalmo/, \/11/. ~, l\'l!. ~. 19:38 gitis. No information is given regarding his treatment and/or follow-up examination. Lyn A. Sedwick, M.D. Transient Visual Loss in Ornithine Transcarbamoylase Deficiency. Snebold NG, Rizzo IF III, Lessell S, Pruett RC. Am J Ophthalmol 1987;104: 407-12 (Oct). [Reprint requests to Dr. S. Lessell, 243 Charles St., Boston, MA 02114.] A 32-year-old man with intermittent decreased vision was found to have ornithine transcarbamoylase deficiency diagnosed by liver biopsy. Exam demonstrated intermittent central scotomata and prolonged latency on visual evoked response. The authors postulate either a toxicity of the optic nerve or geniculocalcarine pathways, but the former seems more likely. Lyn A. Sedwick, M.D. Isolated Trochlear Nerve Palsy Secondary to Cavernous Sinus Meningioma. Slavin ML. Am JOphthalmol 1987;104:433-4 (Oct). [Inquiries to Dr. M. L. Slavin, Department of Ophthalmology, Long Island Jewish Medical Center, New Hyde Park, NY 11042.] A 51-year-old woman developed an isolated right fourth nerve palsy and was found to have a small mass lesion in the cavernous sinus that by magnetic resonance and computerized tomographic scanning resembled meningioma. Nice scans are included to document this unusual cause of a fourth nerve palsy. Lyn A. Sedwick, M.D. Anterior Ischemic Optic Neuropathy: IX. Cup-todisc Ratio and Its Role in Pathogenesis. Beck RW, Servais GE, Hayreh SS. Ophthalmology 1987;94: 1503-8 (Nov). [Reprint requests to Dr. R. W. Beck, Department of Ophthalmology, University of South Florida College of Medicine, 12901 N. 30th St., Tampa, FL 33612.] The cup-to-disk ratio of the optic nerve in the fellow eye of 126 patients with nonarteritic ante- |
