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Show LITERATURE ABSTRACTS 139 was not demonstrated in either patient. These are unusual patients with an atypical history, disease course, and prognosis. Lyn A. Sedwick, M.D. Pontine Infarction: Angiography and Magnetic Resonance Imaging. Baker RS, Carr WA. Surv Ophthalmol1987;32:141-3 (Sept-Oct). [Reprint requests to Dr. R. S. Baker, Department of Ophthalmology, N143 Chandler Medical Center, 800 Rose Street, Lexington, KY 40536-0084.] A 52-year-old man with acute bilateral six nerve paresis and horizontal gaze palsies is presented. Computerized tomographic scanning was negative but magnetic resonance imaging showed extensive decreased signal consistent with infarction in the lower pons. Lyn A. Sedwick, M.D. Orbital Apex Syndrome. Bray WH, Giangiacomo J, Ide CH. Surv Ophthalmol 1987;32:136-40 (SeptOct). [Reprint requests to Dr. J. Giangiacomo, Mason Institute of Ophthalmology, One Hospital Drive, Columbia, MO 65212.] A 16-year-old male with an acutely blind, proptotic left eye with total ophthalmoplegia is discussed. Because of recent anticoagulation, orbital hemorrhage was suspected but clinical course and pathology were subsequently diagnostic of mucormycosis. Causes of orbital apex syndrome are briefly discussed as well as the diagnosis and treatment of mucormycosis. Lyn A. Sedwick, M.D. Visual Recovery From Hypoxic Cortical Blindness During Childhood: Computed Tomographic and Magnetic Resonance Imaging Predictors. Lambert SR, Hoyt CS, Jan JE, Barkovich J, Flodmark O. Arch Ophthalmol 1987;105:1371-7. (Oct). [Reprint requests to Dr. C. S. Hoyt, Department of Ophthalmology, University of California, San Francisco, Room A704, 400 Parnassus Ave., San Francisco, CA 94143.] A review of clinical records of 75 infants and children with a diagnosis of cortical visual impairment was done by these authors who identified 30 patients who experienced hypoxia as the etiology. Computerized tomographic (CT) or magnetic resonance (MR) scans performed at least 1 month after the hypoxic insult were examined by a neuroradiologist who graded injury to the striate and parastriate cortices and the optic radiations. A correlation was found between the severity of CT or MR changes in the optic radiations and final visual function. Although there may be confounding variables (six patients had mild optic atrophy) to the conclusions advanced, this paper has plenty of fruit for thought about these children and why their clinical course is different from that of adults with hypoxic cortical blindess. Lyn A. Sedwick, M.D. Intracranial Oculomotor Nerve Paresis With Anisocoria and Pupillary Parasympathetic Hypersensitivity. Slamovits TL, Miller NR, Burde RM. Am] Ophthalmol1987;104:401-6 (Oct). [Reprint requests to Dr. T. L. Slamovits, Department of Ophthalmology, The Eye and Ear Institute, 203 Lothrop St., Pittsburgh, PA 15213.] Three patients with third nerve palsy involving the pupil caused by intracranial pathology were found to have pilocarpine hypersensitivity without other evidence of tonic pupil (i.e., lightnear dissociation). The authors postulate a "direct, nonsynapsing pathway between the midbrain and the eye" variably present that accounts for these results. Lyn A. Sedwick, M.D. Anterior Segment Ischemia after Recession of Various Recti. Virdi PS, Hayreh SS. Ophthalmology 1987;94:1258-71 (Oct). [Reprint requests to Dr. S. S. Hayreh, Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242.] Rectus muscles were recessed in various combinations with and without posterior ciliary artery occlusion and, in some cases, at two different operations in cynomolgus monkeys. Anterior seg- J C!i" Neuro-ophthalmol, Vol. 8, No.2, 1988 |