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Show 134 LITERATURE ABSTRACTS [ Reprint requests to Dr. H. S. Thompson, Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242.] These companion articles nicely prove some truths that seem to be self- evident: ( a) Normal subjects can have a variable mydriatic response to hydroxyamphetamine but usually with little inter- eye variation, and ( b) hydroxyamphetamine testing in Horner's syndrome usually can distinguish preand postganglionic lesions ( giving enhanced dilation in preganglionic and incomplete dilation in postganglionic lesions compared to the normal pupil). As usual, Dr. Thompson and his colleagues have engineered two more classic studies and important articles in pupil lore. Lyn A. Sedwick, M. D. The Use of Crossed Polarized Filters in the Measurement of the Relative Afferent Pupillary Defect. Rosenberg ML, Oliva A. Am J Ophthalmol 1990; 110: 62- 5 Guly). [ Reprint requests to Dr. M. L. Rosenberg, Department of Neurology, Uniformed Services University of the Health Sciences, 4301 Jones Bridge Road, Bethesda, MD 20814-- 4799.] Instead of neutral- density filters of various strengths, polarizing filters placed in front of each eye were used to quantitate afferent pupillary defect. The authors found this to be a readily reproducible and easily utilized system with greater sensitivity than the use of neutral- density filters. Lyn A. Sedwick, M. D. Artificially Produced Quadrantanopsia in Computed Visual Field Testing. Glovinsky Y, Quigley HA, Bissett RA, Miller NR. Am J Ophthalmol 1990; 110: 90- 1 Guly). [ Inquiries to Dr. Y. Glovinsky, Glaucoma Service, Wilmer Eye Institute, Maumenee B110, 600 N. Wolfe Street, Baltimore, MD 21205.] The authors report a patient referred with bitemporal quadrantanopsia who demonstrated left superior quadrantanopsia on the visual field exam, with all visual fields done on a field analyzer. It appeared to be a functional field defect. An exp. er.. imental subject who tried to reproduce such a defect noted that if no response is given to lights in the desired quadrant at the beginning of the test, then by virtue of the program design larger lights are then shown in this quadrant; this may unwittingly assist an astute malingerer. Lyn A. Sedwick, M. D. Acute Posterior Multifocal Pigment Epitheliopathy and Optic Neuritis in a Family. Wolf MD, Folk JC, Goeken NE. Am J Ophthalmol 1990; 110: 89- 90 Guly). [ Inquiries to Dr. J. C. Folk, Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242.] This study describes three family members, a mother and two adult children, who developed optic neuritis ( mother and son) and acute posterior multifocal pigment epitheliopathy ( daughter) within 5 months of each other. All three had HLA antigens B7 and DR2. Although it is not clear that these individuals lived together or in the same community ( i. e., that they were equally exposed to prevalent infectious diseases), the authors believe these patients' disease represents a spectrum of inflammatory response in genetically susceptible individuals. Lyn A. Sedwick, M. D. Mumps Neuroretinitis in an Adolescent. Foster RE, Lowder CY, Meisler OM, Kosmorsky GS, Baetz- Greenwalt B. Am J OphthalmolI990; 110: 91- 3 Guly). [ Inquiries to Dr. C. Y. Lowder, Department of Ophthalmology, Cleveland Clinic Foundation, 9500 Euclid Avenue, Cleveland, OH 44196- 5042.] A 15- year- old boy developed mumps and neuroretinitis in one eye; these were thought to be related. Because he had been vaccinated at age 31 months, this represents a vaccine failure, estimated to occur in 10 to 25% of the population. Lyn A. Sedwick, M. D. Ganzfield Blankout Occurs in Bowl Perimetry and Is Eliminated by Translucent Occlusion. Fuhr PS, Hershner TA, Daum KM. Arch Ophthalmol 1990; 108: 983- 8 Guly). [ Reprint requests to Dr. P. S. Fuhr, University of Alabama at Birmingham, LITERATURE ABSTRACTS 135 School of OptometryfThe Medical Center, UAB Station, Birmingham, AL 35294.] Ten normal subjects were tested on a Humphrey's machine and 8 reported intermittent darkening of the visual field bowl when one eye was occluded with an opaque patch. The darkening vanished with the use of a translucent patch. Such an occluder was also consistently reported to be more comfortable by the patient, and results of visual testing with the translucent patch showed greater sensitivity and less variance. LYll A. Sedwick, M. D. Protein S Deficiency Associated with Central Retinal Artery Occlusion. Golub BM, Sibony PA, Coller BS, Brook S. Arch Ophthalmol 1990; 108: 918 Uuly). [ No reprint information available.] A 30- year- old man with multiple previous episodes of venous and arterial thrombosis developed a central retinal artery occlusion. He had an inherited protein 5 deficiency believed to be the cause of his occlusive thromboses. Lyn A. Sedwick, M. D. J Clin Neuro- ophthalmol. Vol. 11. No. 2, 1991 |
