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Show LITERATURE ABSTRACTS 227 pletely to antibiotic therapy and another responded incompletely but improved to normal with the addition of corticosteroids orally. Lyn A. Sedwick, M. D. Visual Loss Caused by Rapidly Progressive Intracranial Meningiomas During Pregnancy. Wan WL, Geller JL, Feldon SE, Sadun AA. Ophthalmology 1990; 97: 1~ 21 Oan). [ Reprint requests to Dr. A. A. Sadun, Department of Ophthalmology, University of Southern California School of Medicine, 1355 San Pablo St., Los Angeles, CA 90033.] Three patients with meningiomas ( cavernous sinus, suprasellar, and subfrontal) which produced visual loss during pregnancy are discussed. The authors postulate that these tumors may grow rapidly during pregnancy because of stimulated progesterone receptors in the tumors. Lyn A. Sedwick, M. D. Normal Pupil Size and Anisocoria in Newborn Infants. Roarty JD, Keltner JL. Arch Ophthalmol 1990; 108: 9~ 5 Oan). [ Reprint requests to Dr. J. L. Keltner, Department of Ophthalmology, University of California, Davis, Davis, CA 95616.] Normal infants were photographed to study pupil size. Of 88 photographed, 62 were suitable for study. Of these, 21% ( 13) had a difference in pupil size of at least .5 mm. No pupils had> 1mm difference in size. These pupils were otherwise normal, and the authors conclude that anisocoria of 5- 1.0 mrn in normal infants without any other abnormalities is a finding " of limited significance." Lyn A. Sedwick, M. D. Optic Nerve Damage in Alzheimer's Disease. Sadun AA, Bassi CJ. Ophthalmology 1990; 97: 9- 17 Oan). [ Reprint requests to Dr. A. A. Sadun, Departments of Ophthalmology and Neuro- SurgeIJ:' University of Southern California School of MedIcine, 1355 San Pablo St., Los Angeles, CA 90033.] The authors histologically evaluated optic nerves from 10 patients with Alzheimer's disease and age- matched normal controls. They found specific and nonspecific degeneration especially involving the M- cells, the largest class of retinal ganglion cells, which contribute large caliber fibers to the optic nerve. They speculate that the disproportionate involvement of these cells may help explain the psychophysical complaints and defects seen clinically in these patients. Lyn A. Sedwick, M. D. Ophthalmic Involvement in Aggressive Histiocytosis X. MacCumber MW, Hoffman PN, Wand GS, Epstein JI, Beschorner WE, Green WR. Ophthalmology 1990; 97: 22- 7 Oan). [ Reprint requests to Dr. W. R. Green, Eye Pathology Laboratory, Johns Hopkins Hospital, 600 N. Wolfe St., Baltimore, MD 21205.] A 43- year- old man who had symptoms of pituitary insufficiency and " left rectus palsy" at age 36 is presented. Ultimately, he had involvement of both orbits, the sellae, and many thoracic and abdominal areas with Langerhans cell histiocytosis. Lyn A. Sedwick, M. D. Orbitofrontal Cholesterol Granuloma. McNab AA, Wright JE. Ophthalmology 1990; 97: 2~ 32 Oan). [ Reprint requests to Dr. J. E. Wright, Moorfields Eye Hospital, City Rd., London, ECIV 2PD, England.] The authors reviewed charts from Moorfields Eye Hospital Orbital Clinic and found 27 patients with cholesterol granuloma. In 26 the granuloma had a location in the frontal bone near the lacrimal fossa. In 17, the diagnosis was " confidently made" before surgery, based on clinical and radiographic features. Lyn A. Sedwick, M. D. Functional Scoring of the Field of Binocular Single Vision. Fitzsimons R, White J. Ophthalmology 1990; 97: 33- 5 Oan). [ Reprint requests to Dr. R. Fitzsimons, Sydney Eye Hospital, Sir John Young Crescent, Sydney, Australia.] 1Clin Neur(}-{) phtMlmol, Vol. 10, No. 3, 1990 |
