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Show LITERATURE ABSTRACTS 217 will be up to date in discussing this treatment option with your neurosurgery friends. LYIl A. Sedwick, M. D. Destructive Cysts of the Maxillary Sinus Affecting the Orbit. Kaltreider SA, Dortzbach RK. Arch Ophthalmol 1988; 106: 1398-- 402 ( Oct). [ Reprint requests to Dr. S. A. Kaltreider, P. O. Box 209, Medical College of Virginia Station, Richmond, VA 23298.] Four cases of maxillary sinus mucocele, one with a concurrent retention cyst in the orbit, are presented. Three had prior midface or orbital trauma. Examination demonstrated diplopia, enophthalmos, or proptosis, and three of four cases were improved following surgery. Computerized tomographic scanning nicely demonstrates these lesions, which may occur after a blow- out fracture disturbs the normal architecture of the sinuses. LYIl A. Sedwick, M. D. Orbital Metastases from Prostate Carcinoma. Boldt HC, Nerad JA. Arch Ophthalmo/ 1988; 106: 140~ ( Oct). [ Reprint requests to J. A. Nerad, Department of Ophthalmology, University of Iowa Hospitals and Clinics, Iowa City, IA 52242.] The authors retrospectively identified eight patients with orbital metastases from prostate carcinoma. In three, the primary tumor was unknown at the time of presentation. The patients experienced diplopia, pain, and proptosis. Usually radiologic studies demonstrated an osteoblastic lesion, but osteoclastic lesions and soft tissue masses were also seen. An orbital biopsy was done in two patients but was deemed unnecessary in most, especially if the primary tumor was known and if the orbital lesion was osteoblastic. Although treatment varied, it usually included radiation therapy, and the patients did respond well in the short term. LYIl A. Sedwick, M. D. Brainstem Ocular Motility Defects and AIDS. Hamed LM, Schatz NT, Galetta SL. Am J Ophtha/ mol 1988; 106: 437- 42 ( Oct). [ Reprint requests to Dr. L. M. Hamed, Bascom Palmer Eye Institute, P. O. Box 016880, Miami, FL 33101.] Three patients came to medical attention with acute ocular motility disorders ( some combination of gaze palsy, abducens palsy, and intranuclear ophthalmoplegia), and test results were found to be serologically positive for human immunodeficiency virus. One patient had serologic evidence of syphilis as well. Treatment for presumed toxoplasmosis in the first two patients and for syphilis in the third resulted in clinical improvement. The authors briefly review the relevant neuro- ophthalmic literature and add acquired immunodeficiency syndrome to the list of possible diagnoses in younger patients with brainstem syndromes. LYIl A. Sedwick, M. D. Progressive and Recurrent Nonarteritic Anterior Ischemic Optic Neuropathy. Borchert M, Lessell S. Am J Ophtha/ mol 1988; 106: 443- 9 ( Oct). [ Reprint reqClests to Dr. S. Lessell, Department of Ophthalmology, Massachusetts Eye and Ear Infirmary, 243 Charles St., Boston, MA 02114.] Ten case reports describe patients with recurrent episodes of ischemic optic neuropathy, with further field or acuity loss, progression of visual loss over several weeks following the onset of symptoms, or both. All seemed to have adequate workups, all with sedimentation rates and some with temporal artery biopsies. The authors caution against reassuring patients that visual loss will not be progressive in ischemic optic neuropathy. LYIl A. Sedwick, M. D. A Simple Test for Detection of Monocular Functional Visual Impairment. Savir H, Segal M. Am J Ophtha/ mo/ 1988; 106: 500 ( Oct). [ Inquiries to Dr. H. Savir, Department of Ophthalmology, Hasharon Hospital, Golda Medical Center, Petah- Tiqva 49372, Israel.) The authors recommend using red- green glasses with Ishihara plates to confound functional patients. If the red filter is placed over the eye with alleged poor vision, the Ishihara plates will be visible only to this eye. Correct responses indicate a visual acuity of at least 20/ 400. Another maneuver I elill Neuro- ophthalmol. Vol. 9, No. 3, 1989 |