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Show Analogue! Considerations. Analogical Considerations, 133 down dead From a cannon- hall passingclose beside. have caused the death ofthe patient, and so it his stomach, has not his eneepl‘talon injured? And can there be any alteration of functions has, though in reality, it was neither the cause, nor any part of the cause of his distemper." unaccompanied by some alteration ofstructure? Hitherto we have been obliged to content ourselves with a collective consideration of the grouping, the gradation, and succession of (Nugent on Hydrophobia, 1753, p. 54.)* * The mode, in which this disorder forms, is a question of great interest. Some think that the poison works a slow change in the. part, before. which it is incapable of being symptoms, together with probabilities derived from dissection, as the provisional substitute for deeper insiE;nt into causes. The evidence for the doctrine under examination has ad‘ absorbed, so as to affect the constitution. A case is quoted of double absorption I the bite immediately festering and an axillary gland swelling on the fifth day, with considerable fever, ---but no hydrophobic symptoms occurred till the usual period, otter signs of a second absorption-which signs are said to vanced us not a step further. be pains about the wound rising towards the head or body By a lively and ingenious writer, arguing against the essential inflammatory nature ol' hydrophobia, it is asked: Can any thing that has occurred in dissection " serve to determine whether that inflammation or dryness or whatever else may appear, were originally connected with the hydrophobia; or were only new synfilr toms generated in the course of the disorder? what, in general, can he more different than the cause ot'the distemper, and that of the death, nt'the patient? And yet are not these two Causes frequently eontbunded together, and mistaken one tor the other? OF this let one instance serve instead of a thousand. Stones, passing the ureters, bring on spasmodic strictures-n These, it‘ not relieved in time, bringon an in» tlammntion; this a mortification, and the mat" tilication death. Here, on opening the body: the mortification presents itself and appears to (Tm/25. ofa Soc. 1. 318-19). The, fact seems to me to con~ cur with others inrrendcring it probable that the lymphatics are not the agents in generating hydrophobia. We have here a strong shew of absorption but no disease Whatever like hydrophobia in consequence. If the part must be first changed, why go further than such local change, when you have no evidence of any second absorption; for pains rising to the body or head are none. Do we not see in tetanus and epilepsy, that a local affection without absorption is suffi- cient to agitate the frame through all its fibres, and occasion death? Researches in the line which Dr, Autenrieth tool; m his last dissection, promise the most of all to elucidate the discascz-Multiplex experientin, quze docet hydrophobize seinper quandam titillationem vel dolorem cicatricis przeire, our: ex \‘ulnere tantnm nee nlio modo hydrophobiam oriri probat; ct lymphatica vasa vel conglobatas glanduv lns non ut in alio contagio locali conspitué tumere.--casus in quibus 25, 3a demum die, quin 5st post vulnus acceptum am~ putatio adhuc cum felici successu instituebatur; omnia hacc probant, etsi contagio, variolarum ad instar, tempus quoddam necesse sit ad producendam insignem mutatiouem e vulnere tali vencnato, tuuien nee vasa sanguitcro 11cc lymphatica, nct' eel l ulo 1.- om have |