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Show Journal of Clinical Neuro-ophtiUllmology 13(4): 288-292, 1993. Literature Abstracts Evaluating the Visual Field Effects of Blepharoptosis Using Automated Static Perimetry. Meyer DR, Stern JH, Jarvis JM, Lininger LL. Ophthalmology 1993;100:651-9 (May). [Reprint requests to Dr. D. R. Meyer, Division of Oculoplastic and Orbital Surgery, Department of Ophthalmology, Albany Medical College, One Pinnacle Place, McKown Rd, Albany, NY 12203.] Twenty normal patients with varying degrees of ptosis induced by contoured gold weights placed on an upper eyelid had automated visual field examination with a specially developed fullthreshold static 60° test on the Humphrey field analyzer (using the test axes of 0°,45°,90°, 135°, 180°, 225°, 270°, and 315°). Total test time for one visual field was 10 minutes. Even mild ptosis produced changes in the mean threshold for upper meridians, and severe ptosis produced changes in both upper meridians and less so in 0° and 180° and inferior meridians. The authors feel this setup produces excellent and quick documentation of visual field restriction in patients with ptosis. Lyn A. Sedwick, M.D, Fellow Eye Abnormalities in Acute Unilateral Optic Neuritis. Experience of the Optic Neuritis Treatment Trial. Beck RW, Kupersmith MJ, Cleary PA, Katz B, The Optic Neuritis Study Group. Ophthalmology 1993;100:691-8 (May). [Reprint requests to Dr. R. W. Beck, Department of Ophthalmology, MDC Box 21, University of South Florida College of Medicine, Tampa, FL 33612.] Another nice spinoff article from the Optic Neuritis Treatment Trial, this article looks at visual function in the fellow eye in patients with acute optic neuritis. Abnormalities were found in the fellow eye involving visual field (48%), color vision (21.7%), contrast sensitivity (15.4%), and visual acuity (13.88%), and generally resolved over several months. These findings suggest that fellow eye defects may not represent preexisting optic nerve demyelination in patients with seemingly acute unilateral optic neuritis and should not be used clinically as evidence for a diagnosis of mul- 288 © 1993 Raven Press, Ltd., New York tiple sclerosis. Thomas Slamovits provides a thoughtful discussion immediately following the article. Lyn A, Sedwick, M.D. Orbital Fat Removal. Decompression for Graves Orbitopathy. Trokel 5, Kazim M, Moore S. Ophthalmology 1993;100:674-82 (May). [Reprint requests to Dr. M. Kazim, Edward S. Harkness Eye Institute, 635 West 165 St, New York, NY 10032.] The authors describe their experience with 81 patients with nonactive Graves' orbitopathy who underwent orbital fat removal to alleviate proptosis. Some patients had more than one procedure (17 orbits) and a few (3) had bony decompression as well to "achieve the desired globe position." Some impressive clinical photographs are included. Lyn A. Sedwick, M.D. Operative Complications of Optic Nerve Sheath Decompression. Plotnik JL, Kosmorsky GS. Ophthalmology 1993;100:683-90 (May). [Reprint requests to Dr. G. S. Kosmorsky, Department of Ophthalmology, A31, Cleveland Clinic Foundation, 9500 Euclid Ave, Cleveland, OH 44195-5024.] The authors reviewed 31 patients (38 eyes) with optic nerve sheath decompression surgery for various conditions which had caused visual loss (most for pseudotumor cerebri). Of the 38 eyes, 15 (40%) had complications ranging from relatively minor or tr.ansient ~ocular motili~ disturbance or tonic pupIl) to major (central retinal artery occlusion). The literature regarding complications is reviewed. These authors are to be congratulated for pointing out the very real potential disasters that can be ca~sed by this ~urgery, especially since it is perceived to be Simple and risk-free by so many nonophthalmologists and ophthalmologists alike. Lyn A. Sedwick, M.D. |