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Show Journal of Cliniall Neuro-ophllullmology 7(2): 114-120, 1987. Literature Abstracts Inherited Levator-Medial Rectus Synkinesis. Pang MP, Zweifach PH, Goodwin J. Arch Ophthalmol 1986;104:1489-91 (Oct). [Reprint requests to Dr, J. Goodwin, 1855 Taylor St., Chicago, IL 60612.] Two sisters who each exhibited lid retraction on adduction and/or down-gaze of one eye are discussed. Other family members were believed to be similarly affected but were not examined. The authors postulate that these findings are consistent with a congenital levator-medial rectus synkinesis similar to Marcus-Gunn jaw winking. Lyn A. Sedwick, M.D. Magnetic Resonance Imaging of the Brain in Isolated Optic Neuritis. Johns K, Lavin P, Elliot JH, Partain CL. Arch Ophthalmol 1986;104:1486-8 (Oct). [Reprint requests to Dr. K. Johns, 0-5217, Vanderbilt Medical Center North, Nashville, TN 37232.] Ten patients were studied prospectively who had isolated optic neuritis without historical evidence for demyelination or previous optic neuritis. Bilateral, simultaneous optic neuritis patients were included (three patients, all with abnormal magnetic resonance imaging). After informed consent, these ten patients underwent magnetic resonance scanning (the series was not consecutive, as some patients elected not to participate, for reasons not given). Seven of 10 had abnormal magnetic resonance scans consistent with demyelination, usually with multiple lesions. Follow-up ranged from 2 to 19 months, and only two patients had subsequent symptoms consistent with demyelination during this time. Most patients were scanned between 2 and 30 days after onset of optic neuritis, but one patient, inexplicably, underwent magnetic resonance scann.iI1g...~,mgtlth onset. llte: irti'MOmf ~~cDiG_" ' with optic nellntis have~alJl1ij~~c.' dehce of "mere Wide~dinva cenha! :"l:;,>,<T,ri,illS sysfetnY'One ~'an C'riti;< 114 © 1987 Raven Press, New York conclusion, both because the series is small and biased and because there is some doubt as to whether all "UBOs" (unidentified bright objects) on magnetic resonance imaging represent demye;. lination in the appropriate age group. Lyn A. Sedwick, M.D. Handedness and Esotropia. Lessell S. Arch Ophthalmol1986; 104:1492-4 (Oct). [Reprint requests to Dr. S. Lessell, 243 Charles St., Boston, MA 02114.] Questionnaires were reviewed from 1,083 nonesotropic control subjects and 170 esotropic patients. Patients with accommodative and nonaccommodative esotropia were included but none whose esotropia was secondary to a vision-limiting disorder (Le., cataract). Dr. Lessell found that among control subjects, 6% were left-handed and 7% ambidextrous; in esotropic patients, these percentages increased to 19.6 and 16.8%, respectively. He concludes that "non-right-handedness is probably a marker of anomalous cerebral domi>- , nance, and the disproportion of left-handed and ambidextrous subjects with esotropia may indicate that some persons with esotropia have anomalous brain architecture." Lyn A. Sedwick, M.D. Spasmus Nutans, A Benign Clinical Entity? King RA, Nelson LB, Wagner RS. Arch Ophthalmdl 1986;104:1501-4 (Oct). [Reprint requests to Dr. L. B. Nelson, Department of Pediatric Ophthalmology, Wills Eye Hospital, Ninth and Walnut' Streets, Philadelphia, PA 19107.] The authors are pediatric ophthalmologists who report their experience with 14 patients selecte4 from the pediatric ophthalmology services of Will§; Eye Hospital. Their diagnostic criteria for spasmus> nutans are "(1) acquired unilateral, or asymmetrlt$ bilateral, fine, rapid, horizontal nystagmus; (:Ii |