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Show 294 LITERATURE ABSTRACTS table, which seems unlikely with 2/ 200). The authors make a plea for a central data bank for information on such patients, as Dr. Lessell has advocated in the past, and cautioned against attributing all improvement in patients with traumatic optic neuropathy to surgery or high- dose corticosteroids. Lyn A. Sedwick, M. D. Delayed Radiation Injury to the Retrobulbar Optic Nerves and Chiasm. Clinical Syndrome and Treatment with Hyperbaric Oxygen and Corticosteroids. Roden 0, Bosley TM, Fowble B, Clark J, Savino PJ, Sergott RC, Schatz NJ. Ophthalmology 1990; 97: 346- 51 ( Mar). [ Reprint requests to Dr. T. M. Bosley, Neuro- Ophthalmology Service, Wills Eye Hospital, 9th & Walnut Sts., Philadelphia, PA 19107.] Thirteen patients with radiation optic neuropathy were treated with a combination of corticosteroids and hyperbaric oxygen. No patient's vision improved during therapy, but several lost some during therapy, and several more lost further during follow- up, but usually only a modest amount, e. g., 20/ 40 to 20/ 60. The authors review the literature and nicely catalogue these data in a table giving radiation doses, time to symptoms, and visual acuity. The authors advocate this relatively benign therapy in all such cases, even though their series was uncontrolled, mainly because the natural history of the disease is so devastating. Lyn A. Sedwick, M. D. Acute Homonymous Field Loss: Really a Diagnostic Dilemma. Slavin ML, with comments by Katz B, Hedges T III, Newman S. Serv Ophthalmol 1990; 34: 399- 407 ( Mar- Apr). [ No reprint information given.] This clinical pathologic correlation concerns a 30year- old white woman who developed a partial, lower, homonymous hemianopia. The consultants cover the waterfront of diagnostic possibilities, and all recommend neuroimaging first, with magnetic resonance imaging felt to be slightly better than computerized tomographic scanning given the presentation. She had magnetic resonance scanning consistent wilh demyelinating disease and 4 weeks later had complete resolution of the visual field defect. Lyn A. Sedwick, M. D. Cold- Induced Corneal Edema as an Isolated Feature of Trigeminal Neuropathy. Jacobson OM, Willis RM, Weinstein JM. Am J Ophthalmol 1990; 109: 484-- 6 ( Apr). [ Reprint requests to Dr. D. M. Jacobson, Neura- ophthalmology ( 4F), Marshfield Clinic, 1000 N. Oak Ave., Marshfield, WI 54449.] A patient with traumatic left optic neuropathy, left trigeminal sensory loss, and left VI and VII nerve palsy later developed right visual loss from a carotid- cavernous fistula successfully treated with balloon but 6 months later noticed foggy vision in the right eye on exposure to cold. He was found to have corneal thickening post cold exposure. It was postulated that this finding was secondary to compression of the V cranial nerve from the fistula, which was not noted by the patient until cooler seasons. Lyn A. Sedwick, M. D. Bilateral Choroidal Neonatal Neuroblastoma. Gbis GW, Freeman AI, Pang V, Roloson GJ, Case WF, Ost M, Huntrakoon M, Rothberg PG. Am J OphthalmoI1990; 109: 44S- 9 ( Apr). [ Reprint requests to Dr. G. W. Gbis, 4620 J. C. Nichols Pkwy. # 421, Kansas City, MO 64112.] An infant born with abdominal distension from a liver neuroblastoma was found 2 months later to have bilateral choroidal lesions, sadly confirmed at autopsy 2 weeks later. The nature of the ocular leions, with well- developed rosettes, suggested that all three tumors were primary and that the patient had an inherited neuroblastoma mutation. Lyn A. Sedwick, M. D. Cone Dysfunction in a Subgroup of Patients with Autosomal Dominant Cerebellar Ataxia. Hamilton SR, Chatrian G- E, Mills RP, Kalina RE, Bird TO. Arch OphthalmoI1990; 108: 551- 6 ( Apr). [ Reprint requests to Dr. G- E Chatrian, Division of Electro- |
