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Show 214 LITERATURE ABSTRACTS other causes of uveitis and retinal neovascularization was unrevealing. Lyn A. Sedwick, M. D. Ocular Motility Anomalies in Developmental Misdirection of the Optic Chiasm. McCarty JW, Demer JL, Hovis LA, Nuwer MR. Alii JOphthalmol 1992; 113: 86- 95 ( Jan). [ Reprint requests to Dr. J. L. Demer, Comprehensive Division, Jules Stein Eye Institute, 100 Stein Plaza, UCLA, Los Angeles, CA 90024- 7002.] A 35- year- old nonalbino man with horizontal congenital nystagmus had visual evoked potential testing with full and hemifield stimulation with checkerboard pattern. He was found to have probable nondecussation of nasal retinogeniculate fibers in the chiasm. Ocular motility was studied and suggested an association between the nondecussation and reversed visual tracking and congenital nystagmus. LYIl A. Sedwick, M. D. Hypertropia After Implantation of a Molteno Drainage Device. Munoz M, Parrish R. Alii JOphthalmol 1992; 113: 98- 100 ( Jan). [ Inquired to Dr. M. Munoz, Bascom Palmer Eye Institute, P. O. Box 016880, Miami, FL 33101.] Following implantation of a Molteno tube in the superotemporal quadrant left eye, this patient developed a worsening left hypertropia, which the authors felt arose from a fat adherence syndrome. LYIl A. Sedwick, M. D. Macular Edema After Carotid Endarterectomy in Ocular Ischemic Syndrome. Bierly JR, Dunn JP. Am J OphtlwlmoI1992; 113: 105-- 7 ( Jan). [ Inquiries to Dr. J. R. Bierly, Francis Proctor Foundation, Room 5315, University of California, San Francisco, CA 94143.] A diabetic patient with an ischemic oculopathy but no ipsilateral retinopathy ( by clinical examination or intravenous fluorescein angiography) underwent carotid endarterectomy. Six weeks later, I Cl", Neuro- ophthalmol, Vol. l~, No. 3. 199~ he developed retinopathy and macular ed~ . J in the eye, raising again the question of a " prL,~ ective" effect of significant carotId occluslve disease to the development of diabetic retinopathy. Lyn A. Sedwick, M. D. Visual Recovery in Two Patients After Intravenous Methylprednisolone Treatment of Central Retinal Artery Occlusion Secondary to Giant- Cell Arteritis. Matzkin DC, Slamovits TL, Sachs R, Burde RM. Ophthalmology 1992; 99: 68- 71 ( Jan). [ Reprint requests to Dr. 1. L. Slamovits, Department of Ophthalmology, Montefiore Medical Center/ Albert Einstein College of Medicine, 111 E. 210th St., Bronx, NY 10467.] Two patients with central retinal artery occlusion and temporal arteritis had near complete return of vision following therapy with intravenous methylprednisolone. One had fluctuating visual loss even prior to treatment, but the other had no light perception for 24 hours pretreatment. These truly amazing cases are discussed in detail. LYIl A. Sedwick, M. D. Diagnostic Value and Limitations of Orbital Biopsy in Wegener's Granulomatosis. Kalina PH, Lie ] T, Campbell R], Garrity JA. Ophthalmology 1992; 99: 120- 4 ( Jan). [ Reprint requests to Dr. J. T. Lie, Department of Pathology, Mayo CliniclHilton11, 200 First St., S. W., Rochester, MN 55905.] Mayo Clinic patients from 1976 to 1991 identified from a computer search as haVing a diagnosis of Wegener's granulomatosis form the study group, All patients ( 14) with orbital biopsy were reviewed and those with tissue available for study ( 13) form the cases of this report. The findings on biopsy were variable with granulomatous inflammation present in 62% but multinucleated giant cells in only 31o/ c. Small- vessel vasculitis was seen in 85%. The authors stress the importance of correlating these biopsy findings with extraorbital findings and serological testing ( antineutrophil cvtoplasmic antibody) in order to confirm this dia~: r,:" sis. Lyn A, Sf, \ 1,0. |