| OCR Text |
Show LITERATURE ABSTRACTS 71 of Ophthalmology, regarding optic nerve sheath decompression for progressive nonarteritic ischemic optic neuropathy. The comments are interesting, ranging from scholarly discussion of some possible false scientific reasoning in the rationale behind optic nerve sheath decompression in this disorder. The chief clinical concern was that 2 of the patients might have had lost vision from empty sella syndrome ( in fact, computerized tomographic and magnetic resonance scans ruled this out). The last letter by Dr. Manor is a case report of a patient with progressive nonarteritic ischemic optic neuropathy who improved following an optic nerve sheath decompression. Dr. Sergott et a1. do answer all of the questions raised by these letters. One hopes this lively debate will fuel further study of this type of surgery for progressive nonarteritic ischemic optic neuropathy. LYIl A. Sedwick, M. D. Extracranial Optic Nerve Decompression for Traumatic Optic Neuropathy. Joseph MP, Lessel S, Rizzo J, Momose KJ. Arch Opht/ lalmol 1990; 108: 1091- 3 ( Aug). [ Reprint requests to Dr. M. P. Joseph, Massachusetts Eye and Ear Infirmary, 243 Charles Street, Boston, MA 02114.] A series of 14 patients with indirect optic nerve trauma from head injury were treated 1 to 5 days after injury with optic nerve canal decompression via an external ethmoidectomy. Eleven of 14 had improvement in visual acuity; 11 were 20/ 400 or worse preoperatively and 10 were better than 201 400 post- operatively. Dr. Neil Miller offers very thoughtful positive and negative comments about this study on pages 1086- 87 in this same issue. LYIl A. Sedwick, M. D. Superior Oblique Muscle Dysfunction Following Anterior Ethmoidal Artery Ligation for Epistaxis. Couch JM, Somers ME, Gonzalez C. Arch Ophthalmol 1990; 108: 1110- 3 ( Aug). [ Reprint requests to Dr. J. M. Couch, Midwest Eye Institute, 2700 Hospital Drive., North Kansas City, MO 64116.] Two patients who developed superior oblique muscle underaction following anterior ethmoidal artery ligation for epistaxis are discussed. It was judged likely that surgical trauma or operative he-matoma in the area of the trochlea itself or its periostium is the mechanism for muscle dysfunction. LYIl A. Sedwick, M. D. Relative Afferent Pupillary Defect With Normal Visual Function. Forman S, Behrens MM, Odel IG, Spector RT, Hilal S. Arch Ophthalmol 1990; 108: 1074-- 5 ( Aug). [ Reprint requests to Dr. S. Forman, Department of Ophthalmology, Westchester County Medical Center, Valhalla, NY 10595.] A patient with normal visual function but a left afferent pupillary defect is described. Magnetic resonance scanning showed the presence of a previous hemorrhage in the thalamic- dorsal midbrain area. The authors propose that the lesion involved pupil fibers which had exited the optic tract but had not yet reached pretectal nuclei and were involved in the brachium of the superior colliculus. LYIl A. Sedwick, M. D. Primary Orbital Melanoma Associated With Orbital Melanocytosis. Rice CD, Brown HH. Arch Ophthalmol 1990; 108: 1130- 4 ( Aug). [ Reprint requests to Dr. C. D. Rice, Department of Ophthalmology, University of Arkansas for Medical Sciences, 4301 W. Markham, Mail Slot 523, Little Rock, AR 72205.] A 17- year- old girl developed proptosis during the first trimester of pregnancy and had a surgically removed primary orbital melanoma. This rare orbital tumor and its management are discussed. Lyn A. Sedwick, M. D. Nodular Posterior Scleritis. Brod RD, Saul RF. Arch Ophthalmol 1990; 108: 1170- 1 ( Aug). [ Reprint requests to Dr. R. D. Brod, Department of Ophthalmology, Geisinger Medical Center, Danville, PA 17822.] This nice photo essay gives color fundus photographs, fluorescein angiography, computerized tomographic scanning, and ultrasound pictures of this disorder. Lyn A. Sedwick, M. D. I eliII Neuro- ophlhalmol. Vol. 11. No. 1. 1991 |
