It's Not Just a FAD, (EHR Fatigue Syndrome)

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Identifier walsh_2015_s2_c5-2
Title It's Not Just a FAD, (EHR Fatigue Syndrome)
Creator Jacqueline A. Leavitt; John J. Chen; Diva R. Salomao
Affiliation Mayo Clinic Rochester, MN
Subject Vision loss; Field loss; Fatigue; Intracranial Tumor
History A 29 year-old female nurse, nine months postpartum, presented with an inability to see her computerwell for the past two months. She denied eye pain, diplopia, numbness, tingling or weakness. Therewere no changes in vision in bright vs. dim lighting. She also had a headache at the back of her head fortwo months that was relieved with OTC medications. She denied any events immediately preceding theblurred vision. She also complained of shortness of breath, unexplained weight loss and extreme fatigue,sleeping 10 hours per night and taking naps over her lunch break at work. Workup by her primary caredoctor revealed a normal CXR, ECG with sinus bradycardia and anemia (Hgb 9.6, Hct 28.7). Towardsthe end of her recent pregnancy she was evaluated for polydipsia (drinking up to 9 L /d) and nocturia (6-7 x /night). Water deprivation testing during pregnancy was not possible but sodium of 133 made thediagnosis of diabetes insipidus (DI) unlikely. Symptoms improved after delivery and therefore thepolyuria and polydipsia was attributed to pregnancy. Postpartum she also developed fairly severeanxiety and depression that was managed with sertraline and clonazepam. On examination, bestcorrected visual acuity was 20/30 OU, Ishihara color plates were 11/13 OD, 13/13 OS. Pupils reactedbriskly without an RAPD. Visual fields were full to confrontation. Slit lamp and dilated fundusexamination was unremarkable, including absence of macular abnormalities or optic discpallor. Goldmann fields showed a suggestion of a homonymous field defect, but had variable responsesand the perimetrist noted that she was often falling asleep during the test. Optical coherencetomography showed a normal average retinal nerve fiber layer thickness OU. Lab workup showed anelevated ESR of 70 and an elevated ACE of 62.Tests were performed.
Disease/Diagnosis Langherhans cell histiocytosis involving the chiasm, hypothalamus and sacrum
Date 2015-02
References 1. Writing Group of the Histiocyte Society. Histiocytosis syndromes in children. Lancet. 1:208-209, 1987. 2. Harris NL, Jaffe ES, Diebold J, Flandrin G, Muller-Hermelink HK et al. World Health Organization classification of neoplastic diseases of the hematopoietic and lymphoid tissues: report of the Clinical Advisory Committee meeting-Airlie House, Virginia, November 1997. J Clin Oncol. 17:3835-3849,1999. 3. Howarth DM, Gilchrist GS, Mullan BP, Wiseman GA, Edmonson JH et al. Langerhans cell histiocytosis: diagnosis, natural history, management, and outcome. Cancer. 85:2278-2290, 1999. 4. Grois NG, Favara BE, Mostbeck GH, Prayer D. Central nervous system disease in Langerhans cell histiocytosis. Hematol Oncol Clin North Am. 12:287-305, 1998. 5. Grois N, Flucher-Wolfram B, Heitger A, Mostbeck GH, Hofmann J et al. Diabetes insipidus in Langerhans cell histiocytosis: results from the DAL-HX 83 study. Med Pediatr Oncol. 24:248-256,1995.
Language eng
Format application/pdf
Format Creation Microsoft PowerPoint
Type Text
Source 47th Annual Frank Walsh Society Meeting
Relation is Part of NANOS Annual Meeting Frank B. Walsh Sessions; 2015
Collection Neuro-Ophthalmology Virtual Education Library: Walsh Session Annual Meeting Archives: https://novel.utah.edu/Walsh/
Publisher North American Neuro-Ophthalmology Society
Holding Institution Spencer S. Eccles Health Sciences Library, University of Utah
Rights Management Copyright 2013. For further information regarding the rights to this collection, please visit: https://NOVEL.utah.edu/about/copyright
ARK ark:/87278/s6n0445t
Setname ehsl_novel_fbw
ID 179301
Reference URL https://collections.lib.utah.edu/ark:/87278/s6n0445t