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TitleDescriptionSubjectCollection
201 Ventricular tachycardia with retrograde wenckebachApproximately 50 percent of ventricular tachycardias are associated with AV dissociation. The other 50 percent have retrograde atrial capture. This example shows ventricular tachycardia with retrograde Wenckebach. The retrograde P waves are hard to find, but the arrows are of some help.Wenckebach AV BlockKnowledge Weavers ECG
202 Nonconducted and aberrantly conducted PAC'sIn A the slow sinus rhythm is actually caused by nonconducted PACs hidden in the ST segment. This is confirmed in B where some of the PACs are aberrantly conducted with LBBB, and some PACs are nonconducted.Knowledge Weavers ECG
203 Left anterior fasicular block: frontal plane leadsLeft anterior fascicular block, LAFB, is recognized by left axis deviation of -45 degrees or greater; rS complexes in II, III, aVF; and a small Q wave in I and/or aVL.Knowledge Weavers ECG
204 Right bundle branch block (RBBB)Right bundle branch block (RBBB)Knowledge Weavers ECG
205 All about premature beatsAll about premature beatsKnowledge Weavers ECG
206 Inferoposterior MIInferoposterior MIKnowledge Weavers ECG
207 Neural Network Controlling Wakefulness (Labeled)Brainstem reticular activating system.Royal College of Surgeons in Ireland Illustrations
208 Sino-atrial exit block, type I or wenckebachThis example illustrates 2nd degree sino-atrial exit block. In type 1 S-A block the conduction time between sinus firing and atrial capture progressively prolong, but this cannot be seen on the ECG tracing; type I exit block is inferred if the P-P intervals gradually shorten before the pause and if...Wenckebach AV Block; Ladder DiagramKnowledge Weavers ECG
209 Inferior & Anteroseptal MI + RBBBPathologic Q waves are seen in leads II, III, aVF (inferior MI) and in leads V1-3 (anteroseptal MI). RBBB is recognized by the wide QRS (>0.12s) and the anterior/rightwards orientation of terminal QRS forces. When an anteroseptal MI complicates RBBB (or visa versa) the rSR' complex in V1 (typical ...Knowledge Weavers ECG
210 Atrial flutter with 3:2 conduction ratio: frontal plane leadsNote the subtle bigeminy in the RR intervals. The best way to identify the flutter waves in this example is to imagine what lead III would look like if the QRS complexs disappeared; what remains is a reasonable saw-tooth pattern characteristic of atrial flutter with a flutter rate of about 300 bpm...Knowledge Weavers ECG
211 Accelerated ventricular rhythm with retrograde atrial capture and echo beatsRetrograde atrial captures from an accelerated ventricular focus are occurring with increasing R-P intervals, When the longer R-P occurs, the impulse traversing the AV junction finds a route back to the ventricles, and the result is a ventricular echo.Knowledge Weavers ECG
212 Folate Pool - The Role of B Vitamins in One-Carbon MetabolismThis figure depicts the pathway for folate utilization and the role of vitamins B6 and B12 in the metabolism of methyl-tetrahydrofolate and homocysteine.Folate; FolicinHEAL Open Review Collection
213 ECG components diagram - marquetteECG components diagram - marquetteKnowledge Weavers ECG
214 Postero-lateral MI: Precordial LeadsPostero-lateral MI: Precordial LeadsKnowledge Weavers ECG
215 Indeterminate frontal plane QRS axisIndeterminate frontal plane QRS axisKnowledge Weavers ECG
216 PAC couplet - marquettePAC couplet - marquetteKnowledge Weavers ECG
217 Normal variant: Early repolarizationEarly repolarization, a misnomer, describes a pattern of localized or diffuse ST segment elevation. This is especially seen in leads with prominent R waves. In this example leads I, II, V5 and V6 illustrate the early repolarization pattern. ST segments usually have a concave upwards pattern and ta...Knowledge Weavers ECG
218 AV dissociation by usurpationNormal sinus rhythm is interrupted by an accelerated ventricular rhythm whose rate is slightly faster than the sinus rhythm. Fusion QRS complexes occur whenever the sinus impulse enters the ventricles at the same time the ectopic ventricular focus initiates its depolarization.Knowledge Weavers ECG
219 Long QT mischiefThe long QT ECG has many causes: electrolyte abnormalities including hypo-K, hypo-Mg, and hypo-Ca; drugs including type I antiarrhythmics; CNS injury; and hereditary syndromes. Ventricular arrhythmias are thought to be caused by afterdepolarizations or triggered automaticity.Knowledge Weavers ECG
220 Nonconducted and conducted PAC'sThe pause in this example is the result of a nonconducted PAC, as indicated by the first arrow. The second arrow points to a conducted PAC. The most common cause of an unexpected pause in rhythm is a nonconducted PAC.Knowledge Weavers ECG
221 Atypical LBBB with Q waves in leads I and aVLIn typical LBBB, there are no initial Q waves in leads I, aVL, and V6. If Q waves are present in 2 or more of these leads, myocardial infarction is present.Knowledge Weavers ECG
222 Old inferior MI, PVCs, and atrial fibrillationOld inferior MI, PVCs, and atrial fibrillationKnowledge Weavers ECG
223 Frontal plane QRS axis = -45 degreesFrontal plane QRS axis = -45 degreesKnowledge Weavers ECG
224 Fatty acyl CoA elongation in the endoplasmic reticulumThis shows the overall process of fatty acyl elongation in the endoplasmic reticulum. The process resembles that catalyzed by fatty acyl synthase, but the individual activities appear to be on separate enzymes.Knowledge Weavers Fatty Acids
225 LVH & PVCs: Precordial LeadsLVH & PVCs: Precordial LeadsKnowledge Weavers ECG
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