Title | Creator | Description | Identifier | ||
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1 | MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis Mice | Steven Stasheff; Francisco Nadal-Nicolas; Wei Li; Miriam Bornhorst; Yuan Zhu | To better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no... | 20190319_nanos_sciplatform3_04-video | |
2 | MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis Mice | Steven Stasheff; Francisco Nadal-Nicolas; Wei Li; Miriam Bornhorst; Yuan Zhu | To better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no... | 20190319_nanos_sciplatform3_04_slides | |
3 | MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis Mice | Steven Stasheff; Francisco Nadal-Nicolas; Wei Li; Miriam Bornhorst; Yuan Zhu | To better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no... | 20190319_nanos_sciplatform3_04-abstract |