Preservation of Central Acuity with Macular Infraction: Hypothesis on the Resilience of Foveal Outflow Channels to Retinal Ischemia

Update Item Information
Identifier 19930207_nanos_sciplatform1_05
Title Preservation of Central Acuity with Macular Infraction: Hypothesis on the Resilience of Foveal Outflow Channels to Retinal Ischemia
Creator Joseph F. Rizzo III, MD
Affiliation Massachusetts Eye and Ear
Subject Central Acuity; Diffuse Macular Infraction; Foveal Outflow Channels; Retinal Ischemia
Description The goal of this report is to present hypotheses to explain the mechanisms by which central vision is spared in some instances of diffuse macular infarction. In order to retain central acuity axons from the central-most ganglion cells must survive despite coursing through the densely infracted surrounding macula. Sparing of axons is not known to be a fee feature of neural infarction.
Date 1993-02-07
Language eng
Format application/pdf
Type Text
Source 1993 North American Neuro-Ophthalmology Society Annual Meeting
Relation is Part of NANOS 1993: Platform Presentations (Session 1)
Collection Neuro-ophthalmology Virtual Education Library: NOVEL http://NOVEL.utah.edu
Publisher Spencer S. Eccles Health Sciences Library, University of Utah
Holding Institution North American Neuro-Ophthalmology Association. NANOS Executive Office 5841 Cedar Lake Road, Suite 204, Minneapolis, MN 55416
Rights Management Copyright 1993. For further information regarding the rights to this collection, please visit: https://NOVEL.utah.edu/about/copyright
ARK ark:/87278/s60p45km
Context URL The NANOS Annual Meeting Neuro-Ophthalmology Collection: https://novel.utah.edu/collection/NAM/toc/
Setname ehsl_novel_nam
ID 182916
Reference URL https://collections.lib.utah.edu/ark:/87278/s60p45km
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