The Man with No Face (About Face)

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Identifier walsh_2015_s3_c3-2
Title The Man with No Face (About Face)
Creator Michael Vaphiades; Jennifer Doyle; Lina Nagia; Kevin Bray; Kline Lanning; Glenn Roberson; Adam Quinn; Joel Cure; Katherine Fening
Affiliation (MV) (JD) (LN) (KB) (KL) (AQ) University of Alabama/Ophthalmology Birmingham, AL; (GR) (JC) University of Alabama/Radiology Birmingham, AL; (KF) University of Alabama/Dermatology Birmingham, AL
Subject Basal Cell; Visual Loss; Destructive Process; Head and Neck; Carcinoma
History A 61-year-old man presented with one week history of decreased vision OS. Past medical historyincludes asthma and amblyopia OD, but with complete visual loss OD 18 years prior. He takes nomedications. He is retired but worked for 21 years in social science research. The remarkable thingupon meeting this articulate intelligent individual is he had no face. On exam his visual acuity was NLPOD and HM OS. There was no view of the pupil OD and a poor view OS but there is minimal reactivity.Color vision was non-recordable. Confrontational visual field showed marked constrictionOS. Extraocular muscle exam showed complete restriction OD and a small amount of movement in allcardinal directions OS. External exam OD revealed absence of eyelids, exposed globe, and densecorneal scar with no view into the anterior chamber. External exam OS showed thick, keratinized upperand lower lids temporally and absence of lids medially, and 360 degrees of conjunctival chemosis,diffuse corneal edema with white lesion inferonasally, and a formed AC with poor view. Intraocularpressure was non-measurable OD and 40 mm Hg OS. No view of the fundus OU. Cranial and orbital CTand MRI revealed extensive destruction of soft tissue of the face that abuts and likely invades the dura.The right globe was involved, the left globe appeared intact. The intracranial and prechiasmatic portionof the optic nerves appeared normal. There was no definite evidence of involvement of the cavernoussinuses. However, there was disease surrounding several intracranial nerve roots, which likely representsperineural invasion of these structures. A biopsy was obtained.
Disease/Diagnosis Invasive basal cell carcinoma of the face
Date 2015-02
References 1. Ghanadan A, et al. Different Anatomical Distribution of Basal Cell Carcinoma Subtypes in Iranian Population: Association between Site and Subtype. Ann Dermatol. 2014 ;26:559-63. 2. Menesi W, et al. A reliable frozen section technique for basal cell carcinomas of the head and neck. Can J Plast Surg. 2014;22:179-82. 3. Slutsky JB, Jones EC. Periocular cutaneous malignancies: a review of the literature. Dermatol Surg. 2012;38:552-69. 4. Tan E, et al. Growth of Periocular Basal Cell Carcinoma. Br J Dermatol. 2014 [Epub ahead of print].
Language eng
Format application/pdf
Format Creation Microsoft PowerPoint
Type Text
Source 47th Annual Frank Walsh Society Meeting
Relation is Part of NANOS Annual Meeting Frank B. Walsh Sessions; 2015
Collection Neuro-Ophthalmology Virtual Education Library: Walsh Session Annual Meeting Archives: https://novel.utah.edu/Walsh/
Publisher North American Neuro-Ophthalmology Society
Holding Institution Spencer S. Eccles Health Sciences Library, University of Utah
Rights Management Copyright 2013. For further information regarding the rights to this collection, please visit: https://NOVEL.utah.edu/about/copyright
ARK ark:/87278/s6419tpd
Setname ehsl_novel_fbw
ID 179305
Reference URL https://collections.lib.utah.edu/ark:/87278/s6419tpd
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