Abnormal retinal ganglion cell physiology in mice with neurofibromatosis, optic pathway gliomas, and visual loss

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Identifier 20170404_nanos_posters_182
Title Abnormal retinal ganglion cell physiology in mice with neurofibromatosis, optic pathway gliomas, and visual loss
Creator Steven F. Stasheff
Affiliation Children's National Health System & National Eye Institute, Bethesda, Maryland
Subject Neuro-ophth & systyemic disease ( eg. MS, MG, thyroid), Higher visual functions, Pediatric neuro-ophthalmology, Optic; neuropathy
Description A longstanding mystery complicates the care of patients with a common childhood neurologic disease causing severe visual loss, neurofibromatosis type 1 (NF1). Namely, among the ~20% of young children with NF1 who develop optic pathway gliomas (OPGs), the degree of visual loss or recovery often is not readily predicted by tumor characteristics or treatment response. We investigated whether disruptions of retinal ganglion cell (RGC) physiology might help explain this striking disparity.
Date 2017
Language eng
Format application/pdf
Type Text
Source 2017 North American Neuro-Ophthalmology Society Annual Meeting
Relation is Part of NANOS 2017: Poster Presentations
Collection Neuro-ophthalmology Virtual Education Library: NOVEL http://NOVEL.utah.edu
Publisher Spencer S. Eccles Health Sciences Library, University of Utah
Holding Institution North American Neuro-Ophthalmology Association. NANOS Executive Office 5841 Cedar Lake Road, Suite 204, Minneapolis, MN 55416
Rights Management Copyright 2013. For further information regarding the rights to this collection, please visit: https://NOVEL.utah.edu/about/copyright
ARK ark:/87278/s6h16w2f
Context URL The NANOS Annual Meeting Neuro-Ophthalmology Collection: https://novel.utah.edu/collection/NAM/toc/
Setname ehsl_novel_nam
ID 1272884
Reference URL https://collections.lib.utah.edu/ark:/87278/s6h16w2f
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