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1 MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis MiceSteven Stasheff, Francisco Nadal-Nicolas, Wei Li, Miriam Bornhorst, Yuan ZhuTo better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no...20190319_nanos_sciplatform3_04-video
2 MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis MiceSteven Stasheff, Francisco Nadal-Nicolas, Wei Li, Miriam Bornhorst, Yuan ZhuTo better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no...20190319_nanos_sciplatform3_04-abstract
3 MEK Inhibitor Treatment Promotes Retinal Ganglion Cell Preservation without Preventing Retrobulbar Demyelination in Neurofibromatosis MiceSteven Stasheff, Francisco Nadal-Nicolas, Wei Li, Miriam Bornhorst, Yuan ZhuTo better understand the disparity frequently seen between optic pathway glioma (OPG) characteristics and visual loss among young children with neurofibromatosis type 1 (NF1), we extend our previous findings of anatomic, physiologic, and behavioral abnormalities in an established animal model. We no...20190319_nanos_sciplatform3_04_slides
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