Ceramide-induced disruption of endothelial nitric oxide synthase dimerization in bovine aortic endothelial cells (BAECs) is not secondary to peroxynitrite formation

Update Item Information
Publication Type poster
School or College College of Health
Department Exercise & Sport Science
Creator Kowalski, Christopher Aaron; Deesing, Michole Ann; Deeter, Nicholas B.; Symons, John David
Title Ceramide-induced disruption of endothelial nitric oxide synthase dimerization in bovine aortic endothelial cells (BAECs) is not secondary to peroxynitrite formation
Date 2010-01-20
Description An estimated 23.6 million individuals in the United States have diabetes and of those 90-95% have type II diabetes. Cardiovascular complications (e.g., hypertension and vascular dysfunction) are four-fold more prevalent in patients with type II diabetes. To gain insight into mechanisms responsible for ceramide-induced endothelial dysfunction, BAECs were incubated with palmitate to precipitate endogenous ceramide biosynthesis.
Type Text; Image
Publisher University of Utah
Subject Ceramide-induced disruption; Nitric oxide synthase dimerization; Bovine aortic endothelial cells; BAEC; Peroxynitrite formation; Trapeze Interactive Poster
Subject LCSH Diabetes -- Complications; Ceramides -- Physiological effect
Language eng
Bibliographic Citation Kowalksi, C. A., Deesing, M. A., Deeter, N. B., & Symons, J. D. (2010). Ceramide-induced disruption of endothelial nitric oxide synthase dimerization in bovine aortic endothelial cells (BAECs) is not secondary to peroxynitrite formation.
Rights Management (c) Kowalksi, C. A., Deesing, M. A., Deeter, N. B., & Symons, J. D.
Format Medium application/pdf
Format Extent 477,063 bytes
Identifier ir-main,13134
ARK ark:/87278/s60p1hrv
Setname ir_uspace
ID 707684
Reference URL https://collections.lib.utah.edu/ark:/87278/s60p1hrv
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